Mice lacking uteroglobin are highly susceptible to developing pulmonary fibrosis
Author:
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Reference24 articles.
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3. Identification of the RUSH consensus-binding site by cyclic amplification and selection of targets: demonstration that RUSH mediates the ability of prolactin to augment progesterone-dependent gene expression;Hewetson;Mol. Endocrinol.,2002
4. Severe fibronectin-deposit renal glomerular disease in mice lacking uteroglobin;Zhang;Science,1997
5. Fibrotic disease and the T(H)1/T(H)2 paradigm;Wynn;Nat. Rev. Immunol.,2004
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