A Soluble Sulfogalactosyl Ceramide Mimic Promotes ΔF508 CFTR Escape from Endoplasmic Reticulum Associated Degradation-Reference-Cited by-同舟云学术

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A Soluble Sulfogalactosyl Ceramide Mimic Promotes ΔF508 CFTR Escape from Endoplasmic Reticulum Associated Degradation

Published:2009-04 Issue:4 Volume:16 Page:461-470
ISSN:1074-5521
Container-title:Chemistry & Biology
language:en
Short-container-title:Chemistry & Biology

Author:

Park Hyun-Joo,Mylvaganum Murugesapillai,McPherson Anne,Fewell Sheara W.,Brodsky Jeffrey L.,Lingwood Clifford A.

Publisher

Elsevier BV

Subject

Clinical Biochemistry,Drug Discovery,Pharmacology,Molecular Biology,Molecular Medicine,General Medicine,Biochemistry

Reference62 articles.

1. Processing of CFTR: traversing the cellular maze–how much CFTR needs to go through to avoid cystic fibrosis?;Amaral;Pediatr. Pulmonol.,2005

2. BAG-2 acts as an inhibitor of the chaperone-associated ubiquitin ligase CHIP;Arndt;Mol. Biol. Cell,2005

3. Chaperoning the maturation of the cystic fibrosis transmembrane conductance regulator;Brodsky;Am. J. Physiol. Lung Cell. Mol. Physiol.,2001

4. The protective and destructive roles played by molecular chaperones during ERAD (endoplasmic-reticulum-associated degradation);Brodsky;Biochem. J.,2007

5. Chemical chaperones correct the mutant phenotype of the delta F508 cystic fibrosis transmembrane conductance regulator protein;Brown;Cell Stress Chaperones,1996

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