The Mammalian Malonyl-CoA Synthetase ACSF3 Is Required for Mitochondrial Protein Malonylation and Metabolic Efficiency-Reference-Cited by-同舟云学术

The Mammalian Malonyl-CoA Synthetase ACSF3 Is Required for Mitochondrial Protein Malonylation and Metabolic Efficiency

Published:2017-06 Issue:6 Volume:24 Page:673-684.e4
ISSN:2451-9456
Container-title:Cell Chemical Biology
language:en
Short-container-title:Cell Chemical Biology

Author:

Bowman Caitlyn E.,Rodriguez Susana,Selen Alpergin Ebru S.,Acoba Michelle G.,Zhao Liang,Hartung Thomas,Claypool Steven M.,Watkins Paul A.,Wolfgang Michael J.

Funder

NIH

National Organization of Rare Disease

American Heart Association

Publisher

Elsevier BV

Subject

Clinical Biochemistry,Drug Discovery,Pharmacology,Molecular Biology,Molecular Medicine,Biochemistry

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3. A gene cluster encoding malonyl-CoA decarboxylase (MatA), malonyl-CoA synthetase (MatB) and a putative dicarboxylate carrier protein (MatC) in Rhizobium trifolii – cloning, sequencing, and expression of the enzymes in Escherichia coli;An;Eur. J. Biochem.,1998

4. The clinical significance of small copy number variants in neurodevelopmental disorders;Asadollahi;J. Med. Genet.,2014

5. Age-dependent striatal excitotoxic lesions produced by the endogenous mitochondrial inhibitor malonate;Beal;J. Neurochem.,1993

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