Rapamycin targets STAT3 and impacts c-Myc to suppress tumor growth-Reference-Cited by-同舟云学术

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Rapamycin targets STAT3 and impacts c-Myc to suppress tumor growth

Published:2021-10 Issue: Volume: Page:
ISSN:2451-9456
Container-title:Cell Chemical Biology
language:en
Short-container-title:Cell Chemical Biology

Author:

Sun Le^ORCID,Yan Yu^ORCID,Lv Heng^ORCID,Li Jianlong,Wang Zhiyuan,Wang Kun,Wang Lin^ORCID,Li Yunxia^ORCID,Jiang Hong^ORCID,Zhang Yaoyang^ORCID

Funder

Natural Science Foundation of Shanghai

National Natural Science Foundation of China

Science and Technology Commission of Shanghai Municipality

National Key Research and Development Program of China

Publisher

Elsevier BV

Subject

Clinical Biochemistry,Drug Discovery,Pharmacology,Molecular Biology,Molecular Medicine,Biochemistry

Reference72 articles.

1. Molecular cloning of APRF, a novel IFN-stimulated gene factor 3 p91-related transcription factor involved in the gp130-mediated signaling pathway;Akira;Cell,1994

2. Comprehensive lung injury pathology induced by mTOR inhibitors;Aparicio;Clin. Transl. Oncol.,2009

3. The STAT3 transcription factor is a target for the Myc and riboblastoma proteins on the Cdc25A promoter;Barré;J. Biol. Chem.,2005

4. An alkyne–aspirin chemical reporter for the detection of aspirin-dependent protein modification in living cells;Bateman;J. Am. Chem. Soc.,2013

5. IL-21 signalling via STAT3 primes human naive B cells to respond to IL-2 to enhance their differentiation into plasmablasts;Berglund;Blood,2013

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