The D647N mutation of FGFR1 induces ligand-independent receptor activation-Reference-Cited by-同舟云学术

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The D647N mutation of FGFR1 induces ligand-independent receptor activation

Published:2023-12 Issue:12 Volume:1867 Page:130470
ISSN:0304-4165
Container-title:Biochimica et Biophysica Acta (BBA) - General Subjects
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - General Subjects

Author:

Domenichini Mattia,Ravelli Cosetta,Corsini Michela,Codenotti Silvia,Moreschi Elisa,Gogna Anna,Capoferri Davide,Zizioli Daniela,Bresciani Roberto,Grillo Elisabetta,Mitola Stefania

Funder

Fondazione Umberto Veronesi

Associazione Italiana per la Ricerca sul Cancro

Fondazione Italiana per la Ricerca sul Cancro

Publisher

Elsevier BV

Subject

Molecular Biology,Biochemistry,Biophysics

Reference20 articles.

1. Mechanism of FGF receptor dimerization and activation;Sarabipour;Nat. Commun.,2016

2. FGFR a promising druggable target in cancer: molecular biology and new drugs;Porta;Crit. Rev. Oncol. Hematol.,2017

3. Transformation and stat activation by derivatives of FGFR1, FGFR3, and FGFR4;Hart;Oncogene,2000

4. The precise sequence of FGF receptor autophosphorylation is kinetically driven and is disrupted by oncogenic mutations;Lew;Sci. Signal.,2009

5. Novel potential oncogenic and druggable mutations of FGFRs recur in the kinase domain across cancer types;Grillo;Biochim. Biophys. Acta (BBA) - Mol. Basis Dis.,1868

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1. Necroptosis-related KLRB1 was a potent tumor suppressor and immunotherapy determinant in breast cancer;Heliyon;2024-03

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