Deficiency of neuronal nitric oxide synthase (nNOS) worsens alcohol-induced microencephaly and neuronal loss in developing mice

Author:

Bonthius Daniel J,Tzouras Georgios,Karacay Bahri,Mahoney Jolonda,Hutton Ana,McKim Ross,Pantazis Nicholas J

Publisher

Elsevier BV

Subject

Developmental Biology,Developmental Neuroscience

Reference58 articles.

1. Estimates of nuclear population from microtome sections;Abercrombie;Anat. Rec.,1946

2. Nitric oxide synthase activity in brain regions and spinal cord of mice and rats: kinetic analysis;Barjavel;Pharmacology,1995

3. Ethanol promotes apoptosis in cerebellar granule cells by inhibiting the trophic effect of NMDA;Bhave;J. Neurochem.,1997

4. Blood alcohol concentration and severity of microencephaly in neonatal rats depend on the pattern of alcohol administration;Bonthius;Alcohol,1988

5. D.J. Bonthius, B. Karacay, D. Dai, N.J. Pantazis, FGF-2, NGF and IGF-1, but not BDNF, utilize a nitric oxide pathway to signal neurotrophic and neuroprotective effects against alcohol toxicity in cerebellar granule cell cultures (submitted for publication).

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