Cortical expression of AMPA receptors during postnatal development in a genetic model of absence epilepsy
Author:
Affiliation:
1. Department of AnatomyBrain Health Research Centre, School of Biomedical Sciences, University of OtagoDunedinNew Zealand
Funder
Dean’s Bequest Fund
University of Otago Research Grant
Publisher
Wiley
Subject
Developmental Biology,Developmental Neuroscience
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1016/j.ijdevneu.2018.12.006
Reference39 articles.
1. Alterations in AMPA receptor subunit expression in cortical inhibitory interneurons in the epileptic stargazer mutant mouse
2. Synaptic changes in AMPA receptor subunit expression in cortical parvalbumin interneurons in the stargazer model of absence epilepsy;Adotevi N.K.;Front. Mol. Neurosci.,2017
3. Selective loss of AMPA receptors at corticothalamic synapses in the epileptic stargazer mouse
4. NMDA receptor expression in the thalamus of the stargazer model of absence epilepsy;Barad Z.;Sci. Reports 7,2017
5. Basic developmental rules and their implications for epilepsy in the immature brain;Ben‐Ari Y.;Epileptic Disord.,2006
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2. Parvalbumin Interneuron Dysfunction in Neurological Disorders: Focus on Epilepsy and Alzheimer’s Disease;International Journal of Molecular Sciences;2024-05-19
3. Developmental Inhibitory Changes in the Primary Somatosensory Cortex of the Stargazer Mouse Model of Absence Epilepsy;Biomolecules;2023-01-16
4. Altered GABAA Receptor Expression in the Primary Somatosensory Cortex of a Mouse Model of Genetic Absence Epilepsy;International Journal of Molecular Sciences;2022-12-10
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