Abnormal serotonergic development in a mouse model for the Smith–Lemli–Opitz syndrome: implications for autism-Reference-Cited by-同舟云学术

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Abnormal serotonergic development in a mouse model for the Smith–Lemli–Opitz syndrome: implications for autism

Published:2003-11-26 Issue:8 Volume:21 Page:451-459
ISSN:0736-5748
Container-title:International Journal of Developmental Neuroscience
language:en
Short-container-title:Int. j. dev. neurosci.

Author:

Waage‐Baudet H.¹²,Lauder J.M.¹,Dehart D.B.¹²,Kluckman K.³,Hiller S.³,Tint G.S.⁴,Sulik K.K.¹²

Affiliation:

1. Department of Cell and Developmental BiologyThe University of North CarolinaChapel HillNC27599‐7178USA

2. Bowles Center for Alcohol, StudiesThe University of North CarolinaChapel HillNC27599‐7178USA

3. Department of Cell and Developmental PathologyThe University of North CarolinaChapel HillNC27599‐7178USA

4. VA Hospital EastOrangeNJUSA

Funder

National Alliance for Autism Research

Publisher

Wiley

Subject

Developmental Biology,Developmental Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1016/j.ijdevneu.2003.09.002

Reference70 articles.

1. Review of Fenfluramine in the Treatment of the Developmental Disabilities

2. Fenfluramine Treatment in Infantile Autism

3. Serotonin transporter gene polymorphisms and hyperserotonemia in autistic disorder

4. Abnormal phrenic motoneuron activity and morphology in neonatal monoamine oxidase A‐deficient transgenic mice: possible role of a serotonin excess;Bou‐Flores C.;J. Neurosci.,2000

5. Prenatal development of the serotonin transporter in mouse brain

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