Guanidino compounds inhibit acetylcholinesterase and butyrylcholinesterase activities: Effect neuroprotector of vitamins E plus C-Reference-Cited by-同舟云学术

Guanidino compounds inhibit acetylcholinesterase and butyrylcholinesterase activities: Effect neuroprotector of vitamins E plus C

Published:2010-06-25 Issue:6 Volume:28 Page:465-473
ISSN:0736-5748
Container-title:International Journal of Developmental Neuroscience
language:en
Short-container-title:Int. j. dev. neurosci.

Author:

Delwing‐de Lima Daniela¹²,Wollinger Luiz Felipe³,Casagrande Ana Carolina Mesquita³,Delwing Fábio⁴,Cruz José Geraldo Pereira³,Wyse Angela T.S.⁴,Delwing‐Dal Magro Débora³

Affiliation:

1. Área de Ciências da Saúde, Universidade Comunitária da Região de ChapecóAvenida Senador Attílio Fontana, 591‐ECEP 89809‐000ChapecóSCBrazil

2. Departamento de Ciências da SaúdeUniversidade Regional Integrada do Alto Uruguai e das Missões‐Campus de Frederico WestphalenRua Assis Brasil, 709CEP 98400‐000Frederico WestphalenRSBrazil

3. Departamento de Ciências NaturaisCentro de Ciências Exatas e Naturais, Universidade Regional de BlumenauRua Antônio da Veiga, 140CEP 89010‐971BlumenauSCBrazil

4. Departamento de BioquímicaInstituto de Ciências Básicas da Saúde, Universidade Federal do Rio Grande do SulRua Ramiro Barcelos2600‐AnexoPorto AlegreRSBrazil

Funder

Universidade Regional de Blumenau - Edital interno de apoio à Pesquisa e Extensão and PIBIC/FURB

Publisher

Wiley

Subject

Developmental Biology,Developmental Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1016/j.ijdevneu.2010.06.008

Reference72 articles.

1. Butyrylcholinesterase activity and risk factors for coronary artery disease

2. Acetylcholinesterase promotes the aggregation of amyloid-β-peptide fragments by forming a complex with the growing fibrils 1 1Edited by A. R. Fersht

3. Changes in acetylcholinesterase and butyrylcholinesterase in Alzheimer's disease resemble embryonic development—A study of molecular forms

4. Is there a rationale for the use of acetylcholinesterase inhibitors in the therapy of Alzheimer's disease?

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