Evaluation of neuroprotection by melatonin against adverse effects of prenatal exposure to a nonsteroidal anti‐inflammatory drug during peripheral nerve development

Author:

Keskin Ilknur1,Kaplan Suleyman2,Kalkan Serpil3,Sutcu Mustafa4,Ulkay M. Basak5,Esener O. Burak5

Affiliation:

1. Department of Histology and EmbryologyMedical FacultyMedipol UniversityIstanbulTurkey

2. Department of Histology and EmbryologyMedical FacultyOndokuz Mayis UniversitySamsunTurkey

3. Department of Histology and EmbryologyMedical FacultySelcuk UniversityKonyaTurkey

4. Department of Plastic SurgeryMedical FacultyMedipol UniversityIstanbulTurkey

5. Department of Histology and EmbryologyVeterinary FacultyIstanbul UniversityIstanbulTurkey

Funder

Scientific and Technical Research Council of Selcuk University

Publisher

Wiley

Subject

Developmental Biology,Developmental Neuroscience

Reference48 articles.

1. Age‐dependent cognitive deficits and neuronal apoptosis in cyclooxygenase‐2 transgenic mice;Andreasson K.I.;J. Neurosci.,2001

2. Induction of GDNF mRNA expression by melatonin in rat C6 glioma cells;Armstrong K.J.;Neuroreport,2002

3. Toxicity of non‐steroidal anti‐inflammatory drugs: a review of melatonin and diclofenac sodium association;Aygün D.;Histol. Histopathol.,2012

4. Prenatal exposure of diclofenac sodium affects morphology but not axon number of the median nerve of rats;Ayranci E.;Folia Neuropathol.,2013

5. An efficient stereological sampling approach for quantitative assessment of nerve regeneration;Canan S.;Neuropathol. Appl. Neurobiol.,2008

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