Crosstalk control and limits of physiological c-Jun N-terminal kinase activity for cell viability and neurite stability in differentiated PC12 cells-Reference-Cited by-同舟云学术

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Crosstalk control and limits of physiological c-Jun N-terminal kinase activity for cell viability and neurite stability in differentiated PC12 cells

Published:2017-07 Issue: Volume:82 Page:12-22
ISSN:1044-7431
Container-title:Molecular and Cellular Neuroscience
language:en
Short-container-title:Molecular and Cellular Neuroscience

Author:

Waetzig Vicki,Belzer Milana,Haeusgen Wiebke,Boehm Ruwen,Cascorbi Ingolf,Herdegen Thomas

Funder

Deutsche Forschungsgemeinschaft

German Research Foundation

Publisher

Elsevier BV

Subject

Cell Biology,Cellular and Molecular Neuroscience,Molecular Biology

Reference64 articles.

1. Differentiation of PC12 cells;Adler,2006

2. Distinct roles of c-Jun N-terminal kinase isoforms in neurite initiation and elongation during axonal regeneration;Barnat;J. Neurosci.,2010

3. The mechanism by which MEK/ERK regulates JNK and p38 activity in polyamine depleted IEC-6 cells during apoptosis;Bavaria;Apoptosis,2014

4. SP600125, an anthrapyrazolone inhibitor of Jun N-terminal kinase;Bennett;Proc. Natl. Acad. Sci. U. S. A.,2001

5. Constitutively active cytoplasmic c-Jun N-terminal kinase 1 is a dominant regulator of dendritic architecture: role of microtubule-associated protein 2 as an effector;Bjorkblom;J. Neurosci.,2005

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