Ih, GIRK, and KCNQ/Kv7 channels differently modulate sharp wave - ripples in the dorsal and ventral hippocampus-Reference-Cited by-同舟云学术

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Ih, GIRK, and KCNQ/Kv7 channels differently modulate sharp wave - ripples in the dorsal and ventral hippocampus

Published:2020-09 Issue: Volume:107 Page:103531
ISSN:1044-7431
Container-title:Molecular and Cellular Neuroscience
language:en
Short-container-title:Molecular and Cellular Neuroscience

Author:

Trompoukis George,Rigas Pavlos,Leontiadis Leonidas J.,Papatheodoropoulos Costas

Funder

Hellenic Foundation for Research and Innovation

Publisher

Elsevier BV

Subject

Cell Biology,Cellular and Molecular Neuroscience,Molecular Biology

Reference124 articles.

1. Extracellular calcium controls the expression of two different forms of ripple-like hippocampal oscillations;Aivar;J. Neurosci.,2014

2. Role of KCNQ potassium channels in stress-induced deficit of working memory;Arnsten;Neurobiology of stress,2019

3. Selective participation of somatodendritic HCN channels in inhibitory but not excitatory synaptic integration in neurons of the subthalamic nucleus;Atherton;J. Neurosci.,2010

4. Differential regulation of NMDA receptor-mediated transmission by SK channels underlies dorsal-ventral differences in dynamics of Schaffer collateral synaptic function;Babiec;J. Neurosci.,2017

5. Hippocampal synaptic plasticity, spatial memory and anxiety;Bannerman;Nat. Rev. Neurosci.,2014

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1. Rescue of sharp wave-ripples and prevention of network hyperexcitability in the ventral but not the dorsal hippocampus of a rat model of fragile X syndrome;Frontiers in Cellular Neuroscience;2023-12-01

2. Impairments in hippocampal oscillations accompany the loss of LTP induced by GIRK activity blockade;Neuropharmacology;2023-11

3. Impairments in hippocampal oscillations accompany the loss of LTP induced by GIRK activity blockade;2023-05-05

4. Inhibition of Kv7/M Channel Currents by Fangchinoline;Pharmacology;2022-12-14

5. Gain of function due to increased opening probability by two KCNQ5 pore variants causing developmental and epileptic encephalopathy;Proceedings of the National Academy of Sciences;2022-04-04

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