Differential CaMKII regulation by voltage-gated calcium channels in the striatum-Reference-Cited by-同舟云学术

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Differential CaMKII regulation by voltage-gated calcium channels in the striatum

Published:2015-09 Issue: Volume:68 Page:234-243
ISSN:1044-7431
Container-title:Molecular and Cellular Neuroscience
language:en
Short-container-title:Molecular and Cellular Neuroscience

Author:

Pasek Johanna G.,Wang Xiaohan^ORCID,Colbran Roger J.

Funder

NIH R01

NIH Training Program in Ion Channel and Transporter Biology

National Institute of Mental Health

American Heart Association

Publisher

Elsevier BV

Subject

Cell Biology,Cellular and Molecular Neuroscience,Molecular Biology

Reference116 articles.

1. Combined activation of L-type Ca2+ channels and synaptic transmission is sufficient to induce striatal long-term depression;Adermark;J. Neurosci.,2007

2. Calcium/calmodulin-dependent protein kinase II (CaMKII) inhibition induces neurotoxicity via dysregulation of glutamate/calcium signaling and hyperexcitability;Ashpole;J. Biol. Chem.,2012

3. NMDA receptor subunit composition controls synaptic plasticity by regulating binding to CaMKII;Barria;Neuron,2005

4. Identification of the Ca2+/calmodulin-dependent protein kinase II regulatory phosphorylation site in the alpha-amino-3-hydroxyl-5-methyl-4-isoxazole-propionate-type glutamate receptor;Barria;J. Biol. Chem.,1997

5. Regulatory phosphorylation of AMPA-type glutamate receptors by CaM-KII during long-term potentiation;Barria;Science,1997

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