Remodeling of the Vessel Wall after Copper-Induced Injury Is Highly Attenuated in Mice with a Total Deficiency of Plasminogen Activator Inhibitor-1-Reference-Cited by-同舟云学术

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Remodeling of the Vessel Wall after Copper-Induced Injury Is Highly Attenuated in Mice with a Total Deficiency of Plasminogen Activator Inhibitor-1

Published:2001-01 Issue:1 Volume:158 Page:107-117
ISSN:0002-9440
Container-title:The American Journal of Pathology
language:en
Short-container-title:The American Journal of Pathology

Author:

Ploplis Victoria A.,Cornelissen Ivo,Sandoval-Cooper Mayra J.,Weeks Lisa,Noria Francisco A.,Castellino Francis J.

Funder

National Institutes of Health

American Heart Association

W. M. Keck Foundation

Publisher

Elsevier BV

Subject

Pathology and Forensic Medicine

Reference59 articles.

1. Role of cell-surface lysines in plasminogen binding to cells: identification of α-enolase as a candidate plasminogen receptor;Miles;Biochemistry,1991

2. Plasminogen binds specifically to alpha-enolase on rat neuronal plasma membrane;Hamanoue;J Neurochem,1994

3. Identification of an epitope of alpha-enolase (a candidate plasminogen receptor) by phage display;Arza;Thromb Haemost,1997

4. The urokinase receptor. Protein structure and role in plasminogen activation and cancer invasion;Dana;Fibrinolysis,1994

5. The receptor for urokinase type plasminogen activator polarizes expression of the protease to the leading edge of migrating monocytes and promotes degradation of enzyme inhibitor complexes;Estreicher;J Cell Biol,1995

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