Overexpression of Insulin-Like Growth Factor-1 (IGF-I) Receptor and the Invasiveness of Cultured Keloid Fibroblasts-Reference-Cited by-同舟云学术

Overexpression of Insulin-Like Growth Factor-1 (IGF-I) Receptor and the Invasiveness of Cultured Keloid Fibroblasts

Published:1999-03 Issue:3 Volume:154 Page:883-889
ISSN:0002-9440
Container-title:The American Journal of Pathology
language:en
Short-container-title:The American Journal of Pathology

Author:

Yoshimoto Hiroshi,Ishihara Hiroshi,Ohtsuru Akira,Akino Kozo,Murakami Ryuichi,Kuroda Hiroaki,Namba Hiroyuki,Ito Masahiro,Fujii Tohru,Yamashita Shunichi

Publisher

Elsevier BV

Subject

Pathology and Forensic Medicine

Reference32 articles.

1. Hypertrophic scars, keloids, and contractures: the cellular and molecular basis for therapy;Tredget;Surg Clin North Am,1997

2. Elevated levels of PDGF-α receptors in keloid fibroblasts contribute to an enhanced response to PDGF;Haisa;J Invest Dermatol,1994

3. Keloid fibroblasts exhibit an altered response to TGF-β;Babu;J Invest Dermatol,1992

4. Signal transduction by receptors with tyrosine kinase activity;Ullrich;Cell,1990

5. The protein kinase family: conserved features and deduced phylogeny of the catalytic domains;Hanks;Science,1988

Cited by 71 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. IGF1 and CXCR4 Respectively Related With Inhibited M1 Macrophage Polarization in Keloids;Journal of Craniofacial Surgery;2024-08-15

2. Targeting the Epigenome Reduces Keloid Fibroblast Cell Proliferation, Migration, and Invasion;Journal of Investigative Dermatology;2024-07

3. Comparative Evaluation of Postoperative Scarring with Nasolabial Flap Reconstruction Using Silicone Gel Versus Silicone Gel Sheet: Randomized Controlled Trial;Journal of Maxillofacial and Oral Surgery;2024-06-17

4. The PI3K-Akt-mTOR and Associated Signaling Pathways as Molecular Drivers of Immune-Mediated Inflammatory Skin Diseases: Update on Therapeutic Strategy Using Natural and Synthetic Compounds;Cells;2023-06-20

5. UCHL1 aggravates skin fibrosis through an IGF‐1‐induced Akt/mTOR/HIF‐1α pathway in keloid;The FASEB Journal;2023-05-31