Ablation of angiotensin (1-7) receptor Mas in C57Bl/6 mice causes endothelial dysfunction-Reference-Cited by-同舟云学术

Ablation of angiotensin (1-7) receptor Mas in C57Bl/6 mice causes endothelial dysfunction

Published:2008-11 Issue:6 Volume:2 Page:418-424
ISSN:1933-1711
Container-title:Journal of the American Society of Hypertension
language:en
Short-container-title:Journal of the American Society of Hypertension

Author:

Rabelo Luiza A.,Xu Ping,Todiras Mihail,Sampaio Walkyria O.,Buttgereit Jens,Bader Michael,Santos Robson A.S.,Alenina Natalia

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Internal Medicine

Reference31 articles.

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2. Angiotensin-(1-7) is an endogenous ligand for the G protein-coupled receptor Mas;Santos;Proc Natl Acad Sci U S A,2003

3. Angiotensin-(1-7) prevents development of severe hypertension and end-organ damage in spontaneously hypertensive rats treated with L-NAME;Benter;Am J Physiol,2006

4. Prevention of angiotensin II-induced cardiac remodeling by angiotensin-(1-7);Grobe;Am J Physiol,2007

5. Angiotensin-(1-7) binds to specific receptors on cardiac fibroblasts to initiate antifibrotic and antitrophic effects;Iwata;Am J Physiol,2005

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