Intracellular angiotensin II increases the total potassium current and the resting potential of arterial myocytes from vascular resistance vessels of the rat. Physiological and pathological implications

Author:

De Mello Walmor C.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Internal Medicine

Reference24 articles.

1. Angiotensin II activation of protein kinase C decreases delayed rectifier K+ current in rabbit vascular myocytes;Clement-Chomienne;J Physiol,1996

2. Excitation-contraction coupling and the ultrastructure of smooth muscle;Somlyo;Circ Res,1985

3. Physiological roles and properties of potassium channels in arterial smooth muscle;Nelson;Am J Physiol,1995

4. Phosphorylation by protein kinase A enhances delayed rectifier K+ current in rabbit vascular smooth muscle cells;Aiello;Am J Physiol,1995

5. Protein kinase C-independent inhibition of the Ca!+- activated K+ channel by angiotensin II and endothelin-1;Minami;Biochem Pharmacol,1995

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