Transposition of Duplicated Chromosomal Segment Involving Fused BCR-ABL Gene or ABL Oncogene Alone in Chronic Myelocytic Leukemia and Ph Chromosome-Positive Acute Leukemia with Complex Karyotypes
Author:
Publisher
Elsevier BV
Subject
Cancer Research,Genetics,Molecular Biology
Reference20 articles.
1. Non-random karyotypic evolution in chronic myeloid leukemia;Mitelman;Int J Cancer,1976
2. Philadelphia chromosome breakpoints are clustered within a limited region, bcr, on chromosome 22;Groffen;Cell,1984
3. The chronic myelocytic leukemia-specific P210 protein is the product of the bcr/abl hybrid gene;Ben-Neriah;Science,1986
4. Unique forms of the abl tyrosine kinase distinguish Ph1-positive CML from Ph1-positive ALL;Clark;Science,1987
5. A bcr-v-abl oncogene induces lymphomas in transgenic mice;Hariharan;Mol Cell Biol,1989
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1. Location of the BCR/ABL Fusion Genes on Both Chromosomes 9 in Ph Negative Young CML Patients: An Indian Experience;Indian Journal of Hematology and Blood Transfusion;2014-01-22
2. Emerging stem cell concepts for imatinib-resistant chronic myeloid leukaemia: implications for the biology, management, and therapy of the disease;British Journal of Haematology;2008-08
3. The short arm of chromosome 18 is a recurrent integration site of a supernumerary BCR/ABL gene fusion in chronic myeloid leukemia;Cancer Genetics and Cytogenetics;2007-12
4. New Complex Chromosomal Translocation in Chronic Myeloid Leukaemia: t(9;18;22)(q34;p11;q11);Journal of Biomedicine and Biotechnology;2007
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