Fluorescence In Situ Hybridization Identifies Inversion 16 Masked by t(10;16)(q24;q22), t(7;16)(q21;q22), and t(2;16)(q37;q22) in Three Cases of AML-M4Eo-Reference-Cited by-同舟云学术

Fluorescence In Situ Hybridization Identifies Inversion 16 Masked by t(10;16)(q24;q22), t(7;16)(q21;q22), and t(2;16)(q37;q22) in Three Cases of AML-M4Eo

Published:2000-01 Issue:2 Volume:116 Page:148-152
ISSN:0165-4608
Container-title:Cancer Genetics and Cytogenetics
language:en
Short-container-title:Cancer Genetics and Cytogenetics

Author:

Reddy K.S.,Wang S.,Montgomery P.,Grove W.,Robertson L.E.

Publisher

Elsevier BV

Subject

Cancer Research,Genetics,Molecular Biology

Reference11 articles.

1. Association of an inversion of chromosome 16 with abnormal marrow eosinophils in acute myelomonocytic leukemia;Le Beau;N Engl J Med,1983

2. Cancer Cytogenetics;Heim,1995

3. Molecular pathogenesis of the chromosome 16 inversion in the M4Eo subtype of acute myeloid leukemia;Liu;Blood,1995

4. Catalog of Chromosome Aberrations in Cancer;Mitelman,1994

5. Chromosome 16 and bone-marrow eosinophilia;De la Chapelle;N Engl J Med,1983

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1. Spectral karyotyping and fluorescence in situ hybridization analyses identified a novel three-way translocation involving inversion 16 in therapy-related acute myeloid leukemia M4eo;Cancer Genetics and Cytogenetics;2008-07

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4. Chromosome 16 inversion-associated translocations in acute myeloid leukemia elucidated using a dual-color CBFB DNA probe;Cancer Genetics and Cytogenetics;2002-04

5. A novel variant three-way translocation of inversion 16 in a case of AML-M4eo following low dose methotrexate therapy;Cancer Genetics and Cytogenetics;2001-02