A unique AML1 (CBF2A) rearrangement, t(1;21)(p32;q22), observed in a patient with acute myelomonocytic leukemia
Author:
Publisher
Elsevier BV
Subject
Cancer Research,Genetics,Molecular Biology
Reference61 articles.
1. AML1 and the 8;21 and 3;21 translocations in acute and chronic myeloid leukemia;Nucifora;Blood,1995
2. Isolation of a yeast artificial chromosome spanning the 8;21 translocation breakpoint t(8;21)(q22;q22.3) in acute myelogenous leukemia;Gao;PNAS,1991
3. t(8;21) breakpoints on chromosome 21 in acute myeloid leukemia are clustered within a limited region of a single gene, AML1;Miyoshi;PNAS,1991
4. Identification of breakpoints in t(8;21) acute myelogenous leukemia and isolation of a fusion transcript, AML1/ETO, with similarity to Drosophila segmentation gene, runt;Erickson;Blood,1992
5. Transcriptionally active chimeric gene derived from the fusion of the AML1 gene and a novel gene on chromosome 8 in t(8;21) leukemic cells;Nisson;Cancer Genet Cytogenet,1992
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1. Identification of a novel fusion gene, RUNX1-PRPF38A , in acute myeloid leukemia;International Journal of Laboratory Hematology;2017-03-06
2. Truncated RUNX1 protein generated by a novel t(1;21)(p32;q22) chromosomal translocation impairs the proliferation and differentiation of human hematopoietic progenitors;Oncogene;2015-03-23
3. RUNX1 translocations and fusion genes in malignant hemopathies;Future Oncology;2011-01
4. Additional acquisition of t(1;21)(p32;q22) in a patient relapsing with acute myelogenous leukemia with NUP98-HOXA9;International Journal of Hematology;2008-11-13
5. Genome-wide mapping and characterization of hypomethylated sites in human tissues and breast cancer cell lines;Genome Research;2008-02-06
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