Flusilazole induced developmental toxicity, neurotoxicity, and cardiovascular toxicity via apoptosis and oxidative stress in zebrafish-Reference-Cited by-同舟云学术

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Flusilazole induced developmental toxicity, neurotoxicity, and cardiovascular toxicity via apoptosis and oxidative stress in zebrafish

Published:2024-10 Issue: Volume:284 Page:109993
ISSN:1532-0456
Container-title:Comparative Biochemistry and Physiology Part C: Toxicology & Pharmacology
language:en
Short-container-title:Comparative Biochemistry and Physiology Part C: Toxicology & Pharmacology

Author:

Lee Hojun,An Garam,Lim Whasun,Song Gwonhwa

Publisher

Elsevier BV

Reference77 articles.

1. DNA damage response markers are differentially expressed in BRCA-mutated breast cancers;Aleskandarany;Breast Cancer Res. Treat.,2015

2. Thiobencarb induces phenotypic abnormalities, apoptosis, and cardiovascular toxicity in zebrafish embryos through oxidative stress and inflammation;An;Comp. Biochem. Physiol., Part C: Toxicol. Pharmacol.,2022

3. Heart malformation is an early response to TCDD in embryonic zebrafish;Antkiewicz;Toxicol. Sci.,2005

4. How does p53 induce apoptosis and how does this relate to p53-mediated tumour suppression?;Aubrey;Cell Death Differ.,2018

5. The role of organic matrices in the fate of hydrophobic pesticides: An outdoor stream mesocosm study;Bakanov;Chemosphere,2020

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