FAK-p38 signaling serves as a potential target for reverting matrix stiffness-modulated liver sinusoidal endothelial cell defenestration-Reference-Cited by-同舟云学术

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FAK-p38 signaling serves as a potential target for reverting matrix stiffness-modulated liver sinusoidal endothelial cell defenestration

Published:2024-03 Issue: Volume:305 Page:122462
ISSN:0142-9612
Container-title:Biomaterials
language:en
Short-container-title:Biomaterials

Author:

Zhang Xiaoyu^ORCID,Li Peiwen,Zhou Jin,Zhang Ziliang,Wu Huan,Shu Xinyu,Li Wang,Wu Yi,Du Yu^ORCID,Lü Dongyuan,Lü Shouqin,Li Ning,Long Mian^ORCID

Funder

National Natural Science Foundation of China

Chinese Academy of Sciences

National Key Research and Development Program of China

Publisher

Elsevier BV

Reference64 articles.

1. Fibrosis in chronic liver disease: an update on diagnostic and treatment modalities;Manka;Drugs,2019

2. Liver sinusoidal endothelial cells: physiology and role in liver diseases;Poisson;J. Hepatol.,2017

3. Liver sinusoidal endothelial cells - gatekeepers of hepatic immunity;Shetty;Nat. Rev. Gastroenterol. Hepatol.,2018

4. Role of liver sinusoidal endothelial cells in liver diseases;Gracia-Sancho;Nat. Rev. Gastroenterol. Hepatol.,2021

5. The liver sieve: considerations concerning the structure and function of endothelial fenestrae, the sinusoidal wall and the space of Disse;Wisse;Hepatology,1985

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