Severe deficiencies of IGF-I, IGF-II, IGFBP-3, ALS and paradoxically high-normal bone mass in a child with insulin-resistance syndrome (Rabson-Mendenhall type)-Reference-Cited by-同舟云学术

Severe deficiencies of IGF-I, IGF-II, IGFBP-3, ALS and paradoxically high-normal bone mass in a child with insulin-resistance syndrome (Rabson-Mendenhall type)

Published:2007-10 Issue:5 Volume:17 Page:399-407
ISSN:1096-6374
Container-title:Growth Hormone & IGF Research
language:en
Short-container-title:Growth Hormone & IGF Research

Author:

Fowlkes J.L.,Bunn R.C.,Coleman H.N.,Hall B.,Reid M.C.,Thrailkill K.M.

Publisher

Elsevier BV

Subject

Endocrinology,Endocrinology, Diabetes and Metabolism

Reference45 articles.

1. Lilly Lecture: molecular mechanisms of insulin resistance. Lessons from patients with mutations in the insulin-receptor gene;Taylor;Diabetes,1992

2. Genetically defined forms of diabetes in children;Taylor;J. Clin. Endocrinol. Metab.,1999

3. Mutations in the insulin receptor and their effect on glucose transport;Longo;Trans. Assoc. Am. Physicians,1992

4. Molecular defects of the insulin receptor gene;Accili;Diabetes Metab. Rev.,1995

5. Clinical course of genetic diseases of the insulin receptor (type A and Rabson-Mendenhall syndromes): a 30-year prospective;Musso;Medicine (Baltimore),2004

Cited by 8 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Insulin resistance syndrome presenting with diabetic ketoacidosis — a rare presentation;Journal of Rare Diseases;2023-10-05

2. Incomplete phenotypic presentation in a girl with rare Rabson–Mendenhall syndrome;Acta Diabetologica;2022-11-04

3. Insulin at 100 years – is rebalancing its action key to fighting obesity-related disease?;Disease Models & Mechanisms;2021-11-01

4. Changes in Acid Labile Subunit (ALS) and Insulin-Like Growth Factor 1 (IGF-1) Concentration in Hyperthyroidism before and After Medical Treatment: A Prospective Controlled Study;Endocrinology&Metabolism International Journal;2017-02-22

5. Rabson Mendenhall Syndrome caused by a novel missense mutation;International Journal of Pediatric Endocrinology;2016-11-17