Verapamil- and Cadmium-Resistant Stimulation of Calcium Uptake and Insulin Release by d-Glucose in Depolarised Pancreatic Islets Exposed to Diazoxide
Author:
Publisher
Elsevier BV
Subject
Cell Biology
Reference19 articles.
1. Evidence that glucose can control insulin release independently from its action on ATP-sensitive K+ channels in mouse B cells.
2. Stimulation of Insulin Release by D-Glucose in Depolarized Pancreatic Islets
3. Mechanisms by which glucose can control insulin release independently from its action on adenosine triphosphate-sensitive K+ channels in mouse B cells.
4. Cationic Events Stimulated by D-Glucose in Depolarized Islet Cells
Cited by 4 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Emerging Links between Cadmium Exposure and Insulin Resistance: Human, Animal, and Cell Study Data;Toxics;2020-08-27
2. Assessment of re-aggregated human pancreatic islets for secondary drug screening;British Journal of Pharmacology;2014-05-27
3. Aryl hydrocarbon receptor-mediated effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin on glucose-stimulated insulin secretion in mice;Journal of Applied Toxicology;2009-11
4. Regulation of Insulin Secretion in Islets of Langerhans by Ca2+Channels;Journal of Membrane Biology;2004-07
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