A novel macrolide, solithromycin suppresses mucin overexpression induced by Pseudomonas aeruginosa LPS in airway epithelial cells
Author:
Funder
Ministry of Health, Labour and Welfare
FUJIFILM Toyama Chemical Co., Ltd.
Publisher
Elsevier BV
Subject
Infectious Diseases,Pharmacology (medical),Microbiology (medical)
Reference15 articles.
1. Airway mucus function and dysfunction;Fahy;N Engl J Med,2010
2. LPS-stimulated MUC5AC production involves Rac1-dependent MMP-9 secretion and activation in NCI-H292 cells;Binker;Biochem Biophys Res Commun,2009
3. Azithromycin, clarithromycin and telithromycin inhibit MUC5AC induction by Chlamydophila pneumoniae in airway epithelial cells;Morinaga;Pulm Pharmacol Therapeut,2009
4. Azithromycin inhibits nontypeable Haemophilus influenzae-induced MUC5AC expression and secretion via inhibition of activator protein-1 in human airway epithelial cells;Araki;Eur J Pharmacol,2010
5. Solithromycin, a novel macrolide, does not prolong cardiac repolarization: a randomized, three-way crossover study in healthy subjects;Darpo;J Antimicrob Chemother,2017
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1. Efficacy, safety, and tissue penetration of solithromycin in Japanese patients with otorhinolaryngological infections;Auris Nasus Larynx;2024-04
2. Solithromycin inhibits IL-13-induced goblet cell hyperplasia and MUC5AC, CLCA1, and ANO1 in human bronchial epithelial cells;PeerJ;2023-01-17
3. Nonantimicrobial Actions of Macrolides: Overview and Perspectives for Future Development;Pharmacological Reviews;2021-10
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