NMN Deamidase Delays Wallerian Degeneration and Rescues Axonal Defects Caused by NMNAT2 Deficiency In Vivo-Reference-Cited by-同舟云学术

NMN Deamidase Delays Wallerian Degeneration and Rescues Axonal Defects Caused by NMNAT2 Deficiency In Vivo

Published:2017-03 Issue:6 Volume:27 Page:784-794
ISSN:0960-9822
Container-title:Current Biology
language:en
Short-container-title:Current Biology

Author:

Di Stefano Michele,Loreto Andrea,Orsomando Giuseppe,Mori Valerio,Zamporlini Federica,Hulse Richard P.,Webster Jamie,Donaldson Lucy F.,Gering Martin,Raffaelli Nadia,Coleman Michael P.,Gilley Jonathan,Conforti Laura

Funder

Marie Curie Intra European Fellowship

European Community 7th Framework Programme

Biotechnology and Biological Sciences Research Council (BBSRC)

Medical Research Council (MRC)

Publisher

Elsevier BV

Subject

General Agricultural and Biological Sciences,General Biochemistry, Genetics and Molecular Biology

Reference32 articles.

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2. Wallerian degeneration: an emerging axon death pathway linking injury and disease;Conforti;Nat. Rev. Neurosci.,2014

3. Experiments on the section of glossopharyngeal and hypoglossal nerves of the frog and observations of the alternatives produced thereby in the structure of their primitive fibres;Waller;Philos. Trans. R. Soc. Lond. B Biol. Sci.,1850

4. Endogenous Nmnat2 is an essential survival factor for maintenance of healthy axons;Gilley;PLoS Biol.,2010

5. Subcellular localization determines the stability and axon protective capacity of axon survival factor Nmnat2;Milde;PLoS Biol.,2013

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