Extracellular Reactive Oxygen Species Drive Apoptosis-Induced Proliferation via Drosophila Macrophages-Reference-Cited by-同舟云学术

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Extracellular Reactive Oxygen Species Drive Apoptosis-Induced Proliferation via Drosophila Macrophages

Published:2016-03 Issue:5 Volume:26 Page:575-584
ISSN:0960-9822
Container-title:Current Biology
language:en
Short-container-title:Current Biology

Author:

Fogarty Caitlin E.,Diwanji Neha,Lindblad Jillian L.,Tare Meghana,Amcheslavsky Alla,Makhijani Kalpana,Brückner Katja,Fan Yun,Bergmann Andreas

Publisher

Elsevier BV

Subject

General Agricultural and Biological Sciences,General Biochemistry, Genetics and Molecular Biology

Reference50 articles.

1. Compensatory proliferation and apoptosis-induced proliferation: a need for clarification;Mollereau;Cell Death Differ.,2013

2. Compensatory proliferation induced by cell death in the Drosophila wing disc requires activity of the apical cell death caspase Dronc in a nonapoptotic role;Huh;Curr. Biol.,2004

3. Caspase inhibition during apoptosis causes abnormal signalling and developmental aberrations in Drosophila;Pérez-Garijo;Development,2004

4. Apoptotic cells can induce compensatory cell proliferation through the JNK and the Wingless signaling pathways;Ryoo;Dev. Cell,2004

5. Genetic models of apoptosis-induced proliferation decipher activation of JNK and identify a requirement of EGFR signaling for tissue regenerative responses in Drosophila;Fan;PLoS Genet.,2014

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2. An unscheduled switch to endocycles induces a reversible senescent arrest that impairs growth of the Drosophila wing disc;PLOS Genetics;2024-09-03

3. The Initiator Caspase Dronc Drives Compensatory Proliferation of Apoptosis-Resistant Cells During Epithelial Tissue Regeneration After Ionizing Radiation;2024-09-02

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