Current understanding of phospholipase A2s based on knockout/transgenic mice and human diseases

Author:

Murakami Makoto,Taketomi Yoshitaka

Publisher

Elsevier

Reference178 articles.

1. Progesterone-induced acrosome exocytosis requires sequential involvement of calcium-independent phospholipase A2β (iPLA2β) and group X secreted phospholipase A2 (sPLA2);Abi Nahed;J. Biol. Chem.,2016

2. Inherited human cPLA2α deficiency is associated with impaired eicosanoid biosynthesis, small intestinal ulceration, and platelet dysfunction;Adler;J. Clin. Invest.,2008

3. Group X secreted phospholipase A2 limits the development of atherosclerosis in LDL receptor-null mice;Ait-Oufella;Arterioscler. Thromb. Vasc. Biol.,2013

4. Delivery of siRNA to the mouse brain by systemic injection of targeted exosomes;Alvarez-Erviti;Nat. Biotechnol.,2011

5. Group V secretory phospholipase A2 translocates to the phagosome after zymosan stimulation of mouse peritoneal macrophages and regulates phagocytosis;Balestrieri;J. Biol. Chem.,2006

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