Dephosphorylation of GluN2B C-terminal tyrosine residues does not contribute to acute ethanol inhibition of recombinant NMDA receptors
Author:
Publisher
Elsevier BV
Subject
Behavioral Neuroscience,Neurology,Toxicology,General Medicine,Biochemistry,Health (social science)
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4. Shuffling the deck anew: how NR3 tweaks NMDA receptor function;Cavara;Molecular Neurobiology,2008
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