Tension and Transformation in Pancreas Cancer: Can Phenotype Break Free from the Chrysalis of Genotype?-Reference-Cited by-同舟云学术

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Tension and Transformation in Pancreas Cancer: Can Phenotype Break Free from the Chrysalis of Genotype?

Published:2016-06 Issue:6 Volume:29 Page:780-782
ISSN:1535-6108
Container-title:Cancer Cell
language:en
Short-container-title:Cancer Cell

Author:

DuFort Christopher C.,Hingorani Sunil R.

Funder

NIH/NCI

Lustgarten Foundation

Fred Hutch Solid Tumor Translational Research Program

Publisher

Elsevier BV

Subject

Cancer Research,Cell Biology,Oncology

Reference10 articles.

1. TGF-β-induced nuclear localization of Smad2 and Smad3 in Smad4 null cancer cell lines

2. Preinvasive and invasive ductal pancreatic cancer and its early detection in the mouse

3. Trp53R172H and KrasG12D cooperate to promote chromosomal instability and widely metastatic pancreatic ductal adenocarcinoma in mice

4. KrasG12D and Smad4/Dpc4 Haploinsufficiency Cooperate to Induce Mucinous Cystic Neoplasms and Invasive Adenocarcinoma of the Pancreas

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1. Insights into the mechanisms, regulation, and therapeutic implications of extracellular matrix stiffness in cancer;Bioengineering & Translational Medicine;2024-07-31

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