TIM-3 Regulates CD103+ Dendritic Cell Function and Response to Chemotherapy in Breast Cancer-Reference-Cited by-同舟云学术

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TIM-3 Regulates CD103+ Dendritic Cell Function and Response to Chemotherapy in Breast Cancer

Published:2018-01 Issue:1 Volume:33 Page:60-74.e6
ISSN:1535-6108
Container-title:Cancer Cell
language:en
Short-container-title:Cancer Cell

Author:

de Mingo Pulido Álvaro,Gardner Alycia,Hiebler Shandi,Soliman Hatem,Rugo Hope S.,Krummel Matthew F.,Coussens Lisa M.,Ruffell Brian

Funder

Moffitt Cancer Center

National Cancer Institute

Breast Cancer Research Foundation

Komen Promise

DoD Era of Hope Expansion

NCI/NIH

Moffitt Cancer Center's Shula Breast Cancer

Publisher

Elsevier BV

Subject

Cancer Research,Cell Biology,Oncology

Reference66 articles.

1. Signatures of mutational processes in human cancer;Alexandrov;Nature,2013

2. Promotion of tissue inflammation by the immune receptor Tim-3 expressed on innate immune cells;Anderson;Science,2007

3. Lag-3, Tim-3, and TIGIT: co-inhibitory receptors with specialized functions in immune regulation;Anderson;Immunity,2016

4. Combined blockade of TIM-3 and TIM-4 augments cancer vaccine efficacy against established melanomas;Baghdadi;Cancer Immunol. Immunother.,2013

5. Exhaustion of tumor-specific CD8+ T cells in metastases from melanoma patients;Baitsch;J. Clin. Invest.,2011

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