RasGRP3 Mediates MAPK Pathway Activation in GNAQ Mutant Uveal Melanoma-Reference-Cited by-同舟云学术

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RasGRP3 Mediates MAPK Pathway Activation in GNAQ Mutant Uveal Melanoma

Published:2017-05 Issue:5 Volume:31 Page:685-696.e6
ISSN:1535-6108
Container-title:Cancer Cell
language:en
Short-container-title:Cancer Cell

Author:

Chen Xu,Wu Qiuxia,Depeille Philippe,Chen Peirong,Thornton Sophie,Kalirai Helen,Coupland Sarah E.,Roose Jeroen P.,Bastian Boris C.

Funder

National Cancer Institute

Research to Prevent Blindness

Melanoma Research Alliance

Sandler Program

NIH-NCI Physical Science Oncology Center

NIH

Gabrielle's Angel Foundation

UCSF ACS

UCSF Research Allocation Program

Royal Liverpool University Hospital

Publisher

Elsevier BV

Subject

Cancer Research,Cell Biology,Oncology

Reference43 articles.

1. Activation of RasGRP3 by phosphorylation of Thr-133 is required for B cell receptor-mediated Ras activation;Aiba;Proc. Natl. Acad. Sci. USA,2004

2. Clinical efficacy of a RAF inhibitor needs broad target blockade in BRAF-mutant melanoma;Bollag;Nature,2010

3. Activation of mitogen-activated protein kinase by membrane-targeted Raf chimeras is independent of raft localization;Chen;J. Biol. Chem.,2001

4. Combined PKC and MEK inhibition in uveal melanoma with GNAQ and GNA11 mutations;Chen;Oncogene,2014

5. Ras-dependent mitogen-activated protein kinase activation by G protein-coupled receptors. Convergence of Gi- and Gq-mediated pathways on calcium/calmodulin, Pyk2, and Src kinase;Della Rocca;J. Biol. Chem.,1997

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