RAF1 kinase activity is dispensable for KRAS/p53 mutant lung tumor progression-Reference-Cited by-同舟云学术

RAF1 kinase activity is dispensable for KRAS/p53 mutant lung tumor progression

Published:2021-03 Issue:3 Volume:39 Page:294-296
ISSN:1535-6108
Container-title:Cancer Cell
language:en
Short-container-title:Cancer Cell

Author:

Sanclemente Manuel,Nieto Patricia,Garcia-Alonso Sara,Fernández-García Fernando,Esteban-Burgos Laura,Guerra Carmen,Drosten Matthias,Caleiras Eduardo,Martinez-Torrecuadrada Jorge,Santamaría David,Musteanu Monica,Barbacid Mariano

Publisher

Elsevier BV

Subject

Cancer Research,Cell Biology,Oncology

Reference10 articles.

1. c-Raf, but not B-Raf, is essential for development of K-Ras oncogene-driven non-small cell lung carcinoma;Blasco;Cancer Cell,2011

2. Complete regression of advanced pancreatic ductal adenocarcinomas upon combined inhibition of EGFR and C-RAF;Blasco;Cancer Cell,2019

3. Raf-1 promotes cell survival by antagonizing apoptosis signal-regulating kinase 1 through a MEK-ERK independent mechanism;Chen;Proc. Natl. Acad. Sci. USA,2001

4. Targeting the MAPK pathway in KRAS-driven tumors;Drosten;Cancer Cell,2020

5. Tumor regression and resistance mechanisms upon CDK4 and RAF1 inactivation in KRAS/P53 mutant lung adenocarcinomas;Esteban-Burgos;Proc. Natl. Acad. Sci. USA,2020

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2. Signaling from RAS to RAF: The Molecules and Their Mechanisms;Annual Review of Biochemistry;2024-08-02

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4. Confounding Factors in Targeted Degradation of Short-Lived Proteins;ACS Chemical Biology;2024-07-03

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