Acute poisoning with triethyltin in the rat. Changes in cerebral blood flow, cerebral oxygen consumption, arterial and cerebral venous blood gases
Author:
Publisher
Elsevier BV
Subject
Developmental Neuroscience,Neurology
Reference25 articles.
1. Selective inhibition of glucose oxidation by triethyltin in rat brain in vivo;Cremer;Biochem. J.,1970
2. Reassessment of cerebral capillary changes in acute global ischemia and their relationship to the “no-reflow phenomenon”;Fischer;Stroke,1977
3. Vascular spasm in cat cerebral cortex following ischemia;Hart;Stroke,1978
4. Experimental papilledema: a study of cats and monkeys, intoxicated with triethyltin acetate;Hedges;Neurology,1969
5. Resuscitation of the monkey brain after 1 h complete ischemia. I. Physiological and morphological observations;Hossmann;Brain Res.,1974
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1. Acute Trimethyltin Poisoning Caused by Exposure to Polyvinyl Chloride Production: 8 Cases;The American Journal of the Medical Sciences;2021-07
2. Mechanism in bradycardia induced by Trimethyltin chloride: Inhibition activity and expression of Na+/K+-ATPase and apoptosis in myocardia;The Journal of Toxicological Sciences;2020
3. Ultrastructural studies of cerebral vascular spasm after cardiac arrest-related global cerebral ischemia in rats;Acta Neuropathologica;1995-11
4. Reassessment of a new model of complete cerebral ischemia in rats;Acta Neuropathologica;1991-12
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