γ-Glutamyltransferase activity is unchanged in acutely quinolinate-lesioned rat neostriatum but is elevated in Huntington's disease caudate
Author:
Publisher
Elsevier BV
Subject
Developmental Neuroscience,Neurology
Reference17 articles.
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2. Short- and long-term consequences of intracranial injections of the excitotoxin, quinolinic acid, as evidenced by GFA immunohistochemistry of astrocytes;Bjorklund;Brain Res.,1986
3. Distribution of phosphate-activated glutaminase, pyruvate dehydrogenase and γ-glutamyl transpeptidase in post-mortem brain from Huntington's disease and agonal cases;Butterworth;J. Neurol. Sci.,1985
4. Neostriatal injections: A model for Huntington's chorea;Coyle,1987
5. Developmental changes of endogenous glutamate and γ-glutamyl transferase in cultured cerebral cortical interneurons and cerebellar granule cells, and in mouse cerebral cortex and cerebellum in vivo;Kvamme;Neurochem. Res.,1985
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1. Fetal striatal homotransplantation for Huntington's disease: First two case reports;Neurological Research;1995-08
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