Autocrine stimulation by erythropoietin in transgenic mice results in erythroid proliferation without neoplastic transformation☆☆Supported in part by National Institutes of Health Grants KO8 HL02966 (A.M.) and R29 DK44956 (P.T.C.) and by a Medical Research Foundation of Oregon grant (P.T.C.).-Reference-Cited by-同舟云学术

Autocrine stimulation by erythropoietin in transgenic mice results in erythroid proliferation without neoplastic transformation☆☆Supported in part by National Institutes of Health Grants KO8 HL02966 (A.M.) and R29 DK44956 (P.T.C.) and by a Medical Research Foundation of Oregon grant (P.T.C.).

Published:2003-01 Issue:1 Volume:30 Page:82-89
ISSN:1079-9796
Container-title:Blood Cells, Molecules, and Diseases
language:en
Short-container-title:Blood Cells, Molecules, and Diseases

Author:

Madan A,Lin C,Wang Z,Curtin P.T

Publisher

Elsevier BV

Subject

Cell Biology,Hematology,Molecular Biology,Molecular Medicine

Reference63 articles.

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4. Anemia induces accumulation of erythropoietin mRNA in the kidney and liver;Bondurant;Mol. Cell. Biol.,1986

5. Binding and receptor-mediated endocytosis of erythropoietin in Friend virus-infected erythroid cells;Sawyer;J. Biol. Chem.,1987

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3. Hybrid Model of Erythropoiesis and Leukemia Treatment with Cytosine Arabinoside;SIAM Journal on Applied Mathematics;2011-01

4. Mathematical study of feedback control roles and relevance in stress erythropoiesis;Journal of Theoretical Biology;2010-04

5. The role of erythropoietin receptor expression on tumor cells;Recombinant Human Erythropoietin (rhEPO) in Clinical Oncology;2008