6-Keto-prostaglandin E1 is not equipotent to prostacyclin (PGI2) as an antiaggregatory agent
Author:
Publisher
Elsevier BV
Subject
Endocrinology,Biochemistry
Reference20 articles.
1. An enzyme isolated from arteries transforms prostaglandin endoperoxides to an unstable substance that inhibits platelet aggregation;Moncada;Nature (London),1976
2. Arterial walls are protected against deposition of platelet thrombi by a substance (prostaglandin X) which they make from prostaglandin endoperoxides;Gryglewski;Prostaglandins,1976
3. Modulation of human platelet adenylate cyclase by prostacyclin (PGX);Gorman;Prostaglandins,1977
4. Effects of prostacyclin (PGX) on cyclic AMP concentrations in human platelets;Tateson;Prostaglandins,1977
5. Chemistry of Prostaglandins and Prostacyclins;Axen,1978
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1. Tumor Growth and Metastasis;Platelets;2007
2. Prostanoid receptors and their biological actions;Progress in Lipid Research;1993-01
3. Evidence that circulating 6keto prostaglandin E1 causes the platelet defect of Bartter's syndrome;Prostaglandins, Leukotrienes and Essential Fatty Acids;1990-12
4. PGI2-receptors and molecular mechanisms in platelets and vasculature: State of the art;Pharmacological Research Communications;1987-11
5. Inhibition of adenosine diphosphate-induced intravascular aggregation of rat platelets by 6-oxo-prostaglandin E1;Prostaglandins;1987-09
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