Toxicogenomic study to identify potential signaling alterations related to nasal inflammatory damages induced by diesel exhaust particles in primary human nasal epithelial cells

Author:

Kim Hyun Soo,Kim Hyo Jeong,Kim Nahyun,Song Jae-Jun,Son Bu-Soon,Yang Jun Hyuek,Lee Cheol Min,Park Moo Kyun,Seo Young Rok

Funder

National Research Foundation of Korea

Dongguk University

Publisher

Elsevier BV

Subject

Toxicology,General Medicine

Reference60 articles.

1. Pathogenesis of allergic rhinitis;Baraniuk;J. Allergy Clin. Immunol.,1997

2. Involvement of reactive oxygen species in the metabolic pathways triggered by diesel exhaust particles in human airway epithelial cells;Baulig;Am. J. Phys. Lung Cell. Mol. Phys.,2003

3. Mucin gene expression during differentiation of human airway epithelia in vitro. Muc4 and muc5b are strongly induced;Bernacki;Am. J. Respir. Cell Mol. Biol.,1999

4. Immunolocalization of cytokines in the nasal mucosa of normal and perennial rhinitic subjects. The mast cell as a source of IL-4, IL-5, and IL-6 in human allergic mucosal inflammation;Bradding;J. Immunol.,1993

5. TNF alpha is localized to nasal mucosal mast cells and is released in acute allergic rhinitis;Bradding;Clin. Exp. Allergy,1995

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