DCMQA, a caffeoylquinic acid derivative alleviates NMDA-induced neurotoxicity via modulating GluN2A and GluN2B-containing NMDA receptors in vitro-Reference-Cited by-同舟云学术

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DCMQA, a caffeoylquinic acid derivative alleviates NMDA-induced neurotoxicity via modulating GluN2A and GluN2B-containing NMDA receptors in vitro

Published:2020-09 Issue: Volume:67 Page:104888
ISSN:0887-2333
Container-title:Toxicology in Vitro
language:en
Short-container-title:Toxicology in Vitro

Author:

Yang Yue,Gao Huan,Liu Wenwu,Jiang Xiaowen,Shen Zexu,Li Xiang,Ren Tianshu,Xu Zihua,Cheng Gang,Zhao Qingchun

Funder

National Natural Science Foundation of China

NSFC

Publisher

Elsevier BV

Subject

Toxicology,General Medicine

Reference61 articles.

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2. Excitotoxic neuroprotection and vulnerability with CaMKII inhibition;Ashpole;Mol. Cell. Neurosci.,2011

3. Calcium/calmodulin-dependent protein kinase II (CaMKII) inhibition induces neurotoxicity via dysregulation of glutamate/calcium signaling and gyperexcitability;Ashpole;J. Biol. Chem.,2012

4. Apoptotic mechanisms involved in neurodegenerative diseases: experimental and therapeutic approaches;Camins;Method. Find. Exp. Clin.,2008

5. Salidroside attenuates glutamate-induced apoptotic cell death in primary cultured hippocampal neurons of rats;Chen;Brain Res.,2008

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