Response to Stegmann S, et al. [J. Clin. Virol. 47 (1) (2010) 79–81]
Author:
Publisher
Elsevier BV
Subject
Infectious Diseases,Virology
Reference17 articles.
1. Foscarnet as salvage therapy in HIV-2-infected patient with antiretroviral treatment failure;Stegmann;J Clin Virol,2009
2. Foscarnet salvage therapy efficacy is associated with the presence of thymidine-associated mutations (TAMs) in HIV-infected patients;Charpentier;J Clin Virol,2008
3. Reverse transcriptases from human immunodeficiency virus type 1 (HIV-1), HIV-2, and simian immunodeficiency virus (SIVMAC) are susceptible to inhibition by foscarnet and 3′-azido-3′-deoxythymidine triphosphate;Vrang;Antimicrob Agents Chemother,1988
4. Acyclovir is activated into a HIV-1 reverse transcriptase inhibitor in herpesvirus-infected human tissues;Lisco;Cell Host Microbe,2008
5. The antiherpetic drug acyclovir inhibits HIV replication and selects the V75I reverse transcriptase multidrug resistance mutation;McMahon;J Biol Chem,2008
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