Hepatotoxicity induced in rats by chronic exposure to F–53B, an emerging replacement of perfluorooctane sulfonate (PFOS)-Reference-Cited by-同舟云学术

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Hepatotoxicity induced in rats by chronic exposure to F–53B, an emerging replacement of perfluorooctane sulfonate (PFOS)

Published:2024-04 Issue: Volume:346 Page:123544
ISSN:0269-7491
Container-title:Environmental Pollution
language:en
Short-container-title:Environmental Pollution

Author:

Li Xiaohan,Zhang Qian,Wang Aiqing,Shan Shan,Wang Xueying,Wang Yarong,Wan Jianmei,Ning Ping,Hong Chengjiao,Tian Hailin,Zhao Yun

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Reference47 articles.

1. Triglyceride metabolism in the liver;Alves-Bezerra;Compr. Physiol.,2018

2. Modeling the global fate and transport of perfluorooctane sulfonate (PFOS) and precursor compounds in relation to temporal trends in wildlife exposure;Armitage;Environ. Sci. Technol.,2009

3. Triglyceride, small, dense low-density lipoprotein, and the atherogenic lipoprotein phenotype;Austin;Curr. Atherosclerosis Rep.,2000

4. Liver fibrosis;Aydin;Turk. J. Gastroenterol.,2018

5. Developmental toxicity of perfluorooctanesulfonate (PFOS) and its chlorinated polyfluoroalkyl ether sulfonate alternative F-53B in the domestic chicken;Briels;Environ. Sci. Technol.,2018

Cited by 4 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Molecular designing of potential environmentally friendly PFAS based on deep learning and generative models;Science of The Total Environment;2024-11

2. Impact of the novel chlorinated polyfluorinated ether sulfonate, F-53B, on gill structure and reproductive toxicity in zebrafish;Aquatic Toxicology;2024-10

3. Exacerbated interfacial impacts of nanoplastics and 6:2 chlorinated polyfluorinated ether sulfonate by natural organic matter in adult zebrafish: Evidence through histopathology, gut microbiota, and transcriptomic analysis;Journal of Hazardous Materials;2024-09

4. PFOS Exposure Promotes Hepatotoxicity in Quails by Exacerbating Oxidative Stress and Inflammation-Induced Apoptosis through Activating TLR4/MyD88/NF-κb Signaling;ACS Omega;2024-05-31

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