OTA induces intestinal epithelial barrier dysfunction and tight junction disruption in IPEC-J2 cells through ROS/Ca2+-mediated MLCK activation-Reference-Cited by-同舟云学术

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OTA induces intestinal epithelial barrier dysfunction and tight junction disruption in IPEC-J2 cells through ROS/Ca2+-mediated MLCK activation

Published:2018-11 Issue: Volume:242 Page:106-112
ISSN:0269-7491
Container-title:Environmental Pollution
language:en
Short-container-title:Environmental Pollution

Author:

Wang Hong,Zhai Nianhui,Chen Ying,Fu Chongyang,Huang Kehe

Funder

National Natural Science Foundation of China

National Key R & D Program

Postgraduate Research & Practice Innovation Program of Jiangsu Province

Priority Academic Program Development of Jiangsu Higher Education Institutions

Publisher

Elsevier BV

Subject

Health, Toxicology and Mutagenesis,Pollution,Toxicology,General Medicine

Reference46 articles.

1. Ochratoxin A and its metabolites in urines of German adults-An assessment of variables in biomarker analysis;Ali;Toxicol. Lett.,2017

2. Intestinal barrier function in health and gastrointestinal disease;Camilleri;Neurogastroenterology Motil.,2012

3. Gut microbiota, enteroendocrine functions and metabolism;Cani;Curr. Opin. Pharmacol.,2013

4. Pro-inflammatory effects of ochratoxin A on nasal epithelial cells;Cremer;Eur. Arch. Oto-Rhino-Laryngol.,2012

5. Myosin light chain kinase: pulling the strings of epithelial tight junction function;Cunningham,2012

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