Rh1 high activity binding peptides inhibit high percentages of Plasmodium falciparum FVO strain invasion-Reference-Cited by-同舟云学术

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Rh1 high activity binding peptides inhibit high percentages of Plasmodium falciparum FVO strain invasion

Published:2013-04 Issue:14 Volume:31 Page:1830-1837
ISSN:0264-410X
Container-title:Vaccine
language:en
Short-container-title:Vaccine

Author:

Arévalo-Pinzón Gabriela,Curtidor Hernando,Muñoz Marina,Suarez Diana,Patarroyo Manuel A.,Patarroyo Manuel E.

Publisher

Elsevier BV

Subject

Infectious Diseases,Public Health, Environmental and Occupational Health,General Veterinary,General Immunology and Microbiology,Molecular Medicine

Reference54 articles.

1. Invasion of red blood cells by malaria parasites;Cowman;Cell,2006

2. Cooperativity between Plasmodium falciparum adhesive proteins for invasion into erythrocytes;DeSimone;Mol Microbiol,2009

3. Phenotypic variation of Plasmodium falciparum merozoite proteins directs receptor targeting for invasion of human erythrocytes;Duraisingh;EMBO J,2003

4. Molecular mechanism for switching of P. falciparum invasion pathways into human erythrocytes;Stubbs;Science,2005

5. Erythrocyte entry by malarial parasites. A moving junction between erythrocyte and parasite;Aikawa;J Cell Biol,1978

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1. Plasmodium falciparum rhoptry neck protein 4 has conserved regions mediating interactions with receptors on human erythrocytes and hepatocyte membrane;International Journal of Medical Microbiology;2023-05

2. The Cellular and Molecular Interaction Between Erythrocytes and Plasmodium falciparum Merozoites;Frontiers in Cellular and Infection Microbiology;2022-03-31

3. Sexual forms obtained in a continuous in vitro cultured Colombian strain of Plasmodium falciparum (FCB2);Malaria Journal;2020-02-03

4. Receptor-ligand and parasite protein-protein interactions inPlasmodium vivax: Analysing rhoptry neck proteins 2 and 4;Cellular Microbiology;2018-03-26

5. Plasmodium vivax ligand-receptor interaction: PvAMA-1 domain I contains the minimal regions for specific interaction with CD71+ reticulocytes;Scientific Reports;2017-08-30

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