Antisense oligonucleotides ameliorate kidney dysfunction in podocyte-specific APOL1 risk variant mice
Author:
Funder
National Institutes of Health
Publisher
Elsevier BV
Subject
Drug Discovery,Pharmacology,Genetics,Molecular Biology,Molecular Medicine
Reference48 articles.
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2. APOL1 and cardiovascular disease: a story in evolution;Bajaj;Arterioscler. Thromb. Vasc. Biol.,2017
3. APOL1: the balance imposed by infection, selection, and kidney disease;Beckerman;Trends Mol. Med.,2018
4. APOL1 variants: from parasites to kidney function to cardiovascular disease;Reiner;Arterioscler. Thromb. Vasc. Biol.,2016
5. Missense mutations in the APOL1 gene are highly associated with end stage kidney disease risk previously attributed to the MYH9 gene;Tzur;Hum. Genet.,2010
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1. Splice-Modulating Antisense Oligonucleotides as Therapeutics for Inherited Metabolic Diseases;BioDrugs;2024-01-22
2. Reducing reductionism: addressing risk for atherosclerotic cardiovascular disease by apolipoprotein proteomics;European Heart Journal;2023-06-02
3. APOL1 and APOL1-Associated Kidney Disease: A Common Disease, an Unusual Disease Gene – Proceedings of the Henry Shavelle Professorship;Glomerular Diseases;2023-01-25
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