P2Y12 inhibitor clopidogrel inhibits renal fibrosis by blocking macrophage-to-myofibroblast transition-Reference-Cited by-同舟云学术

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P2Y12 inhibitor clopidogrel inhibits renal fibrosis by blocking macrophage-to-myofibroblast transition

Published:2022-09 Issue:9 Volume:30 Page:3017-3033
ISSN:1525-0016
Container-title:Molecular Therapy
language:en
Short-container-title:Molecular Therapy

Author:

Chen Junzhe,Tang Ying,Zhong Yu,Wei Biao,Huang Xiao-Ru,Tang Patrick Ming-Kuen,Xu Anping,Lan Hui-yao

Publisher

Elsevier BV

Subject

Drug Discovery,Pharmacology,Genetics,Molecular Biology,Molecular Medicine

Reference58 articles.

1. Action plan for determining and monitoring the prevalence of chronic kidney disease;Coresh;Kidney Int. Suppl.,2017

2. The myofibroblast matrix: implications for tissue repair and fibrosis;Klingberg;J. Pathol.,2013

3. Pericytes and perivascular fibroblasts are the primary source of collagen-producing cells in obstructive fibrosis of the kidney;Lin;Am. J. Pathol.,2008

4. Endothelial-myofibroblast transition contributes to the early development of diabetic renal interstitial fibrosis in streptozotocin-induced diabetic mice;Li;Am. J. Pathol.,2009

5. Tubular epithelial-myofibroblast transdifferentiation in progressive tubulointerstitial fibrosis in 5/6 nephrectomized rats;Ng;Kidney Int.,1998

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1. Novel glucomannan-like polysaccharide from Lycium barbarum L. ameliorates renal fibrosis via blocking macrophage-to-myofibroblasts transition;International Journal of Biological Macromolecules;2024-10

2. Clopidogrel protects against gentamicin-induced nephrotoxicity through targeting oxidative stress, apoptosis, and coagulation pathways;Naunyn-Schmiedeberg's Archives of Pharmacology;2024-09-05

3. Metabolic reaction fluxes as amplifiers and buffers of risk alleles for coronary artery disease;2024-08-20

4. Clopidogrel protect against gentamicin-induced nephrotoxicity through targeting oxidative stress, apoptosis, and coagulation pathways;2024-08-02

5. The role of macrophages in fibrosis of chronic kidney disease;Biomedicine & Pharmacotherapy;2024-08

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