Subchronic inhalation of coal dust particulate matter 10 induces bronchoalveolar hyperplasia and decreases MUC5AC expression in male Wistar rats

Author:

Kania Nia,Setiawan Bambang,Widjadjanto Edi,Nurdiana Nurdiana,Aris Widodo M.,Chandra Kusuma H.M.S.

Publisher

Elsevier BV

Subject

Cell Biology,Toxicology,General Medicine,Pathology and Forensic Medicine

Reference21 articles.

1. Examination of lung toxicity, oxidant/antioxidant status and effect of erdosteine in rats kept in coal mine ambience;Armutcu;Environ Toxicol Pharmacol,2007

2. Role of iron in inactivation of epidermal growth factor receptor after asbestos treatment of human lung and pleural target cells;Baldys;Am J Respir Cell Mol Biol,2004

3. Genotype and phenotype in susceptibility to coal workers's pneumoconiosis. The use of cytokine in perspective;Born;Eur Respir J,2001

4. The epidermal growth factor (EGF);Cohen;Cancer,1983

5. Hydroxyl radical generation by coal mine dust: possible implication to coal workers’pneumoconiosis;Dalal;Free Rad Biol Med,1995

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