Progression of renal fibrosis in congenital CKD model rats with reduced number of nephrons-Reference-Cited by-同舟云学术

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Progression of renal fibrosis in congenital CKD model rats with reduced number of nephrons

Published:2017-06 Issue:5 Volume:69 Page:245-258
ISSN:0940-2993
Container-title:Experimental and Toxicologic Pathology
language:en
Short-container-title:Experimental and Toxicologic Pathology

Author:

Yasuda Hidenori,Tochigi Yuki,Katayama Kentaro,Suzuki Hiroetsu

Funder

Kidney Foundation, Japan

Ministry of Education, Culture, Sports, Science, and Technology of Japan

Publisher

Elsevier BV

Subject

Cell Biology,Toxicology,General Medicine,Pathology and Forensic Medicine

Reference53 articles.

1. Transforming growth factor-beta1 is up-regulated by podocytes in response to excess intraglomerular passage of proteins: a central pathway in progressive glomerulosclerosis;Abbate;Am. J. Pathol.,2002

2. Human nephron number: implications for health and disease;Bertram;Pediatr. Nephrol.,2011

3. Role of platelet-derived growth factors in mouse development;Betsholtz;Int. J. Dev. Biol,1995

4. PDGF-D inhibition by CR002 ameliorates tubulointerstitial fibrosis following experimental glomerulonephritis;Boor;Nephrol. Dial. Transplant.,2007

5. Regular moderate exercise reduces advanced glycation and ameliorates early diabetic nephropathy in obese Zucker rats;Boor;Metabolism,2009

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