Protein kinase C inhibitors arrest the C6 glioma cell cycle at a mid-G1 phase restriction point: Implications for the antiproliferative action of valproate-Reference-Cited by-同舟云学术

Protein kinase C inhibitors arrest the C6 glioma cell cycle at a mid-G1 phase restriction point: Implications for the antiproliferative action of valproate

Published:1997-01 Issue:1 Volume:12 Page:9-14
ISSN:0887-2333
Container-title:Toxicology in Vitro
language:en
Short-container-title:Toxicology in Vitro

Author:

O'Brien E,Regan C.M

Publisher

Elsevier BV

Subject

Toxicology,General Medicine

Reference28 articles.

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1. Valproate activates phosphodiesterase-mediated cAMP degradation: relevance to C6 glioma G1 phase progression;Neurotoxicology and Teratology;2004-02

2. Differential enantioselective effects of pentyl-4-yn-valproate on spatial learning in the rat, and neurite outgrowth and cyclin D3 expression in vitro;J NEUROCHEM;2004

3. Differential enantioselective effects of pentyl-4-yn-valproate on spatial learning in the rat, and neurite outgrowth and cyclin D3 expression in vitro;Journal of Neurochemistry;2003-12-22

4. Antiproliferative action of valproate is associated with aberrant expression and nuclear translocation of cyclin D3 during the C6 glioma G1 phase;Journal of Neurochemistry;2002-09-18

5. Antiproliferative actions of inhalational anesthetics: comparisons to the valproate teratogen;International Journal of Developmental Neuroscience;2000-02