Nicotine suppresses inflammatory factors in HBE16 airway epithelial cells after exposure to cigarette smoke extract and lipopolysaccharide-Reference-Cited by-同舟云学术

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Nicotine suppresses inflammatory factors in HBE16 airway epithelial cells after exposure to cigarette smoke extract and lipopolysaccharide

Published:2010-12 Issue:6 Volume:156 Page:326-334
ISSN:1931-5244
Container-title:Translational Research
language:en
Short-container-title:Translational Research

Author:

Li Qi,Zhou Xiangdong,Kolosov Victor P.,Perelman Juliy M.

Publisher

Elsevier BV

Subject

Biochemistry, medical,Public Health, Environmental and Occupational Health,General Medicine,Physiology (medical)

Reference40 articles.

1. Emerging tobacco hazards in China: 2. Early mortality results from a prospective study;Niu;BMJ,1998

2. The impact of tobacco on lung health in China;Zhang;Respirology,2003

3. Regulation of mucin genes in chronic inflammatory airway diseases;Voynow;Am J Respir Cell Mol Biol,2006

4. The role of airway secretions in COPD: pathophysiology, epidemiology and pharmacotherapeutic options;Roqers;COPD,2005

5. Nicotinic acetylcholine receptor alpha7 subunit is an essential regulator of inflammation;Wang;Nature,2003

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1. The controversial effect of smoking and nicotine in SARS-CoV-2 infection;Allergy, Asthma & Clinical Immunology;2023-06-01

2. Aerosolized nicotine from e-cigarettes alters gene expression, increases lung protein permeability, and impairs viral clearance in murine influenza infection;Frontiers in Immunology;2023-03-14

3. Prolonged, physiologically relevant nicotine concentrations in the airways of smokers;American Journal of Physiology-Lung Cellular and Molecular Physiology;2023-01-01

4. Differential responses to e-cig generated aerosols from humectants and different forms of nicotine in epithelial cells from nonsmokers and smokers;American Journal of Physiology-Lung Cellular and Molecular Physiology;2021-06-01

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