Deficiency of Smad3 results in enhanced inducible nitric oxide synthase–mediated hypotension in lipopolysaccharide-induced endotoxemia

Author:

Lv Kai-Yang,Zhong Qi-Sheng,Liu Xue-Feng,Zhu Shi-Hui,Xiao Shi-Chu,Wang Guang-Yi,Ma Bing,Xia Zhao-Fan

Publisher

Elsevier BV

Subject

Surgery

Reference29 articles.

1. Pathophysiology of septic shock and implications for therapy;Barron;Clin Pharm,1993

2. Nitric oxide-derived nitrate anion contributes to endotoxic shock and multiple organ injury/dysfunction;Yang;Crit Care Med,2002

3. L-arginine: nitric oxide pathway in endotoxemia and human septic shock;Gómez-Jiménez;Crit Care Med,1995

4. Resistance to endotoxic shock in endothelial nitric-oxide synthase (eNOS) knock-out mice: a pro-inflammatory role for eNOS-derived no in vivo;Connelly;J Biol Chem,2005

5. In vivo inhibition of nuclear factor-kappa B activation prevents inducible nitric oxide synthase expression and systemic hypotension in a rat model of septic shock;Liu;J Immunol,1997

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